Gut Bacteria Connection to Leanness and Obesity

Gut Bacteria Connection to Leanness and Obesity

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Gut Bacteria Connection to Leanness and Obesity

It is interesting to learn that intestinal bacteria play a significant role in determining people’s obesity and leanness. Among high percentage of people struggling to fight obesity, their causes are known to be unlucky genes, unhealthy nutrition and to some extent the genetic composition. Recently, it is evident that the gut bacteria play an extra role in assisting in breaking down tough plant fibre. They are essential in altering how fat is stored, how people respond to a hormone that makes them full or hungry as well as hoe the glucose the glucose level is balance in the bold. From the birth moment, wrong microbes’ combination helps to set diabetes and obesity stage from the birth period. The researchers are optimistic in learning the mechanics of healthy and wrong microbe mix that will significantly play a vital role in controlling obesity as well as weight.

It is through gene sequencing methodologies that researchers revealed that most microbiotas reside in the mouth and large intestines. However, the genital tract and the skin also harbours a good number of these diverse bacteria. Some are acquired during birth from the mother’s birth canal while others are gained from the environment as one grows. Research showed that within lean people the microbe species are like a rainforest while in obese people’s gut the microbiome is diverse. The microbes significantly breakdowns the plant fibres and starches into smaller molecules that are readily used by the body as an energy source. Gordon and his colleague research using baby rodents proved the point right as those that received bacteria from the twin obese and with diverse microbe population grew fat than the ones with bacteria from the lean twins (Dao et al. 2015). When sharing the same cage, all the mouse emerged thin as the rodents with microbes from obese human had picked gut bacteria from to other lean rodents through consumption of their faecal matter.

Furthermore, the children born through the caesarian section as well as those feed on artificial milk are more prone to obesity than those that are naturally born through the vaginal tract and feeds on the breast milk. They are also disposed to asthma, allergies, celiac diseases and eczema. It is because they do not acquire bacteria contained in the mother’s genital tract as well as they do not get the immune acquired through breastfeeding that is vital in fighting the susceptible diseases at early ages. Potential treatments for the same has been established where babies born through caesarian section are directly cleansed with a gauze cloth fastened with mother’s vaginal fluids as well as the inhabitant microbiota (Tilg & Moschen 2014). Transfer of faecal matter from lean people to the obese can significantly be used to deal with the disorder leading to weight loss, even though it is not encouraged as it is risky and imprecise. Gordon suggested that enriching food with the required microbes as well as nutrients appropriate to establish them will significantly be a sound treatment for obesity.

In consideration of the past study, obesity is significantly lead by positive energy balance as well as overeating. It is well that the disease is related to the poor balancing of nutrition. For instance, these people tend overfeeding energy sources such as fats and carbohydrates and being inactive when it comes carrying out body exercise (Boulangé et al. 2016). The further research done by the scientist regarding obesity is significant as the addition of the gut bacteria can work well in many people than before.

The article is ethically based are coming up with sound solutions regarding obesity are healthy to the society. Researchers such as Gordon Robert Karp and Dominquez-Bello has played a crucial role in the field of medicine. I agree with them regarding the research and techniques they applied in finding obesity medication. Eventually, the scientists work in treating obesity is useful and will significantly eradicate the disease in the society.


Boulangé, C. L., Neves, A. L., Chilloux, J., Nicholson, J. K., & Dumas, M. E. (2016). Impact of the gut microbiota on inflammation, obesity, and metabolic disease. Genome medicine, 8(1), 42.

Dao, M. C., Everard, A., Aron-Wisnewsky, J., Sokolovska, N., Prifti, E., Verger, E. O., … & Dumas, M. E. (2015). Akkermansia muciniphila and improved metabolic health during a dietary intervention in obesity: relationship with gut microbiome richness and ecology. Gut, gutjnl-2014.

Tilg, H., & Moschen, A. R. (2014). Microbiota and diabetes: an evolving relationship. Gut, 63(9), 1513-1521.